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Series of systemic lupus erythematosus clinical significance of coagulation test


st1 so that LAC can interfere with arachidonic acid metabolism, reduce prostacyclin (PGI) synthesis. PGI to reduce the concentration of circulating platelet aggregation may be an important reason for increased . Also, according to results of this paper we consider , Ⅷ R: Ag and PFn elevated levels may be further increased platelet adhesion and aggregation ,[link widoczny dla zalogowanych], in platelet thrombosis. Thromboembolic complications in 3 cases of SLE patients, plasma fibronectin ,[link widoczny dla zalogowanych], Ⅷ factor related antigen and platelet aggregation were significantly higher than normal ,[link widoczny dla zalogowanych], indicating that these three may be related to thrombosis have a certain relationship. Major anticoagulant and coagulation - fibrinolysis indicators observed that SLE patients with AT11: Ag, AT Ⅲ: C and Plg were not significantly changed ,[link widoczny dla zalogowanych], suggesting that hypercoagulable state in SLE ​​patients is not due to coagulation and fibrinolytic system due to changes in system function . So we think that SLE patients because of enhanced platelet aggregation ,[link widoczny dla zalogowanych], secondary procoagulant (PFn, Ⅷ R: AgJ ~ species such as the increase in the hypercoagulable state . the presence of lupus anticoagulant in patients with SLE may be associated with recurrent miscarriage or premature birth , thrombocytopenia And thrombotic complications Gui plug is closely related. and its anticoagulant and fibrinolytic system in no significant change.